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Suppressing hemangio- & lymphangiogenesis by splice MO against VEGFR2 (KDR)

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jonmoulton
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Topic Started by jonmoulton
on 9/21/2012 8:37 AM   
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Uehara H, Cho Y, Simonis J, Cahoon J, Archer B, Luo L, Das SK, Singh N, Ambati J, Ambati BK.  Dual suppression of hemangiogenesis and lymphangiogenesis by splice-shifting morpholinos targeting vascular endothelial growth factor receptor 2 (KDR).  FASEB J, 2012;[Epub ahead of print] doi:10.1096/fj.12-213835

Jon D. Moulton, Ph.D. Gene Tools, LLC www.gene-tools.com


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sarmed
Iraq

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Posted By sarmed
on 10/5/2012 13:03 PM   
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hi would you help me out with this please>>>>>>>>

http://www.scientistsolutions.com/t24456-over_expression+the+pepck_c+enzyme+in+the+human+skeletal+muscle_.html



jonmoulton
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Posted By jonmoulton
on 11/13/2012 11:44 AM   
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It's an interesting question, Sarmed, but I don't want to speculate about human treatments for performance enhancement.

Jon D. Moulton, Ph.D. Gene Tools, LLC www.gene-tools.com



sarmed
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Posted By sarmed
on 11/13/2012 12:08 PM   
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 ok .... so I was wondering :

1) Viral transfection is the best type for in vivo transfection as far as effectivity so what's gonna happen for the letiviral or adeno-virus after transfection is done and the targeted gene has been knocked-out?

2) Which one of the other methods comes in the second place

3) what is  the best way  to active immunize against some gene ... better than risking knock it out while we can only make the immune syste  produce some antibodies against it????

 



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